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October 5, 2010

N-Trivia

N-Trivia


Scoliosis

Posted: 04 Oct 2010 08:17 PM PDT


scoliosis picture 300x224 Scoliosis

Scoliosis

Scoliosis

  • A lateral curvature of the spine, may be found in thoracic, lumbar, or thoracolumbar spinal segment.
  • The curve may be convex to the right (more common in lumbar curves) or to the left (more common in lumbar curves).
  • Rotation of the vertebral column around its axis occurs and may cause rib cage deformity.
  • It is often associated with kyposis (humpback) and lordocis (swayback).

Etiology And Pathophysiology

  1. Idiopathic scoliosis – exact etiology is unknown. Accounts for 65% of cases. Possible causes include genetic factors, vertebral growth abnormality. Classified into three groups based on age at time of diagnosis.
    • Infantile – birth to age 3.
    • Juvenile – presentation between age 11 and 17.
  2. Congenital scoliosis – exact etiology unknown; represented as malformation of one or more vertebral bodies that results in asymmetric growth.
    • Type I – failure of vertebral body formation e.g. isolated hemivertebra, wedged vertebra, multiple wedged vertebrae, and multiple hemivertebrae.
    • Type II – failure of segmentation e.g. unilateral unsegmented bar, bilateral block vertebra.
    • Commonly associated with other congenital anomalies.
  3. Paralytic or musculoskeletal scoliosis – develops several months after symmetrical paralysis of the trunk muscles from polio, cerebral palsy, or muscular dystrophy.
  4. Neuromascular scoliosis – child has a definite neuromascular condition that directly contributes to the deformity.
  5. Additional but less common causes of scoliosis are osteopathic conditions, such as fractures, bone disease, arthritic conditions, and infections.
  6. Miscellaneous factors that can cause scoliosis include spinal irradiation, endocrine disoders, postthoracotomy, and nerve root irritation.
  7. As the deformity progresses, changes in the thoracic cage increase. Respiratory and cardiovascular compromise can occur in cases of severe progression.

Assessment

  1. Poor posture, uneven shoulder height.
  2. One hip more prominent than the other.
  3. Scapular prominence.
  4. Uneven waist line or hemline
  5. Spinal curve observable or palpable on both upright and bent forward.
  6. Back pain may be present but is not a routine finding in idiopathic scoliosis.
  7. Leg length discrepancy.

Nursing Diagnosis

  • Disturbed body image related to negative feelings about spinal deformity and appearance in brace.
  • Risk for impaired skin integrity related to mechanical irritation to brace.
  • Risk for injury related to postoperative complications.

Diagnostic Evaluation

  1. X-ray of the spine in the upright position, preferably on one long 36-inch cassette, show characteristic curvature.
  2. MRI, myelograms, or CT scan with three dimensional reconstruction may be indicated for children with severe curvatures who have a known or suspected spinal column anomaly, before management decisions are made.
  3. Pulmonary function tests for compromised respiratory status.
  4. Evaluate for renal abnormalities in children with congenital scoliosis.

Nursing Interventions

  1. Prepare the child for casting or immobilization procedure by showing materials to be used and describing procedure in age-appropriate terms.
  2. Promote comfort with proper fit of brace or cast.
  3. Provide opportunity for the child to express fears and ask questions about deformity and brace wear.
  4. Assess skin integrity under and around the brace or cast frequently.
  5. Provide good skin care to prevent breakdown around any pressure areas.
  6. Instruct the patient to examine brace daily for signs of loosening or breakage.
  7. Instruct patient to wear cotton shirt under brace to avoid rubbing.
  8. Instruct about which previous activities can be continued in the brace.
  9. Provide a peer support person when possible so the child can associate positive outcomes and experiences from others.
image from healthguide.howstuffworks.com

Related posts:

  1. Correction Of Scoliosis
  2. Osteoarthritis
  3. Cerebrospinal Fluid (CSF) Analysis

Osteoarthritis

Posted: 04 Oct 2010 08:16 PM PDT


osteoarthritis 300x240 Osteoarthritis

Osteoarthritis

Osteoarthritis

  • The most common form of arthritis.
  • It causes the deterioration of the joint cartilage and formation of reactive new bone at the margins and subchondral areas of the joint.
  • This chronic degeneration results from a breakdown of chondrocytes, most often in the hips and knees.
  • Osteoarthritis occurs equally in both sexes after age 40.
  • The earliest symptoms appear in middle age and progress with advancing age.
  • Depending on the site and severity of joint involvement, disability can range from minor limitation of the fingers to near immobility in persons with hip or knee disease.
  • Progression rates vary; joints may remain stable for years in the early stage of deterioration.

Etiology And Pathophysiology

  1. Changes in articular cartilage occur first; later, secondary soft tissue changes may occur.
  2. Progressive wear and tear on cartilage leads to thinning of joint surface and ulceration into bone.
  3. Leads to inflammation of the joint and increased blood flow and hypertrophy of subchondral bone.
  4. New cartilage and bone formation at joint margins results in osteophytosis, altering the size and shape of the bone.
  5. Generally affects adults ages 50 to 90; equal to males and females.
  6. Cause is unknown, but aging and obesity are contributing factors. Previous trauma cause secondary osteoarthritis.

Assessment

  1. Deep, aching pain; pronounced after weight bearing or exercise, usually relieved by rest.
  2. Joint swelling or deformity.
  3. Joint stiffness on awakening, usually lasting less than 30 minutes.
  4. Hard nodes on distal or proximal interphalangeal joints on the fingers; known as Heberden's nodes when present on distal interphalangeal joints and Bouchard's nodes when present on proximal interphalangeal joints.

Nursing Diagnosis

  • Chronic pain related to joint deterioration.

Diagnostic Evaluation

  1. X-rays of affected joints show joint space narrowing and sclerosis.
  2. Radionuclide imaging (bone scan) may show increased uptake in affected bones.
  3. Synovial fluid analysis will differentiate osteoarthritis from rheumatoid arthritis by low cell count.

Nursing Interventions

  1. Provide rest for involved joints. Excessive use aggravates the symptoms and accelerates degeneration.
  2. Advise the patient to avoid activities that precipitate pain.
  3. Apply heat as directed to relieve muscle pain and stiffness.
  4. Teach the patient correct posture and body mechanics.
  5. Advise the patient to sleep with rolled terry cloth towel under the neck to relieve cervical pain.
  6. Provide patient with crutches, braces, or cane when indicated to reduce-weight bearing stress on hips and knees.
  7. Encourage patient to wear corrective shoes and metatarsal support for foot disorders.
  8. Encourage patient to lose weight to decrease stress on weight-bearing joints.
  9. Teach the patient range-of-motion exercises to maintain join mobility.
  10. Refer patient to physical and occupational therapy.

Complications

  • Osteoarthritis may cause flexion contractures, subluxation and deformity, ankylosis, bony cysts, gross bony overgrowth, central cord syndrome, nerve root compression, and cauda equine syndrome.
image from healthguide.howstuffworks.com

Related posts:

  1. Hodgkin's Disease
  2. What is Prostate Cancer
  3. Gout

Traumatic Brain Injury

Posted: 04 Oct 2010 08:14 PM PDT


Traumatic Brain Injury

  • Also known as head injury.
  • Is the disruption of normal brain function due to trauma-related injury resulting in compromised neurologic function resulting in focal or diffuse symptoms.
  • Motor vehicle accidents are the most common etiology of injury.

Etiology And Pathophysiology

Types of Traumatic Brain Injury

  1. Concussion – transient interruption in brain activity; no constructural injury noted on radiographics.
  2. Cerebral contusion – bruising of brain with associated swelling.
  3. Intracerebral hematoma – bleeding into the brain tissue commonly associated with edema.
  4. Epidural hematoma – blood between the inner table of the skull and dura.
  5. Subdural hematoma – blood between the dura and arachnoid caused by bleeding commonly associated with additional brain injury.
  6. Diffuse axonal injury – axonal tears within the white matter of the brain.

Assessment

  1. Disturbance in level of consciousness from slightly drowsy to unconscious.
  2. Headache, vertigo, agitation, and restlessness.
  3. Cerebrospinal fluid leakage at ears and nose, which may indicate skull fracture.
  4. Contusions about eyes and ears indicating skull fractures.
  5. Irregular respirations
  6. Cognitive deficit
  7. Pupillary abnormality
  8. Sudden onset of neurologic deficits
  9. Otorrhea indicating posterior fossa skull fracture
  10. Rhinorrhea indicating anterior fossa skul fracture.

Nursing Diagnosis

  • Risk for injury related to complications of head injury.
  • Acute pain related to altered brain or skull tissue.

Diagnostic Evaluation

  1. CT identifies and localizes lesions, cerebral edema, and bleeding.
  2. Skull and cervical spine X-ray identify fracture and displacement.
  3. Complete blood count, coagulation profile, electrolyte levels, serum osmolarity, arterial blood gases, and other laboratory tests monitor for complications.
  4. Neuropsychological test during rehabilitation phase determine cognitive deficits.

Nursing Interventions

  1. Maintain ICP monitoring, as indicated, and report abnormalities.
  2. Maintain patent airway; assist with intubation and ventilatory assistance is needed.
  3. Turn the patient every 2 hours and encourage coughing and deep breathing.
  4. Apply firm pressure over puncture site for subdural trap, and observe for drainage and dressing.
  5. Suction the patient as needed.
  6. Institute measures to prevent increased ICP or other neurovascular compromise.
  7. Feed the patient as soon as possible after a head injury and administer histamine-2 blockers to prevent gastric ulceration and hemorrhage from gastric acid hypersecretion.
  8. If the patient is unable to swallow, provide enteral feedings after bowel sounds have returned.
  9. Elevate the head of the bed after feedings, and check residuals to prevent aspiration.
  10. Monitor respiratory rate, depth, and pattern of respirations.

Complications

  • Infections
  • Increased intracranial pressure
  • Posttraumatic seizure disorder
  • Permanent neurologic deficits
  • Persistent sympathetic storming
  • SIADH
  • Death

Related posts:

  1. Spinal Cord Injury (SCI)
  2. Nursing Care Plan – Spinal Cord Injury
  3. Arteriovenous Malformation (AVM)

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